Title | REST Final-Exon-Truncating Mutations Cause Hereditary Gingival Fibromatosis. |
Publication Type | Journal Article |
Year of Publication | 2017 |
Authors | Bayram, Y, White, JJ, Elcioglu, N, Cho, MT, Zadeh, N, Gedikbasi, A, Palanduz, S, Ozturk, S, Cefle, K, Kasapcopur, O, Akdemir, ZCoban, Pehlivan, D, Begtrup, A, Carvalho, CMB, Paine, ISophie, Mentes, A, Bektas-Kayhan, K, Karaca, E, Jhangiani, SN, Muzny, DM, Gibbs, RA, Lupski, JR |
Corporate Authors | Baylor-Hopkins Center for Mendelian Genomics |
Journal | Am J Hum Genet |
Volume | 101 |
Issue | 1 |
Pagination | 149-156 |
Date Published | 2017 Jul 06 |
ISSN | 1537-6605 |
Keywords | Adolescent, Base Sequence, Chromosome Segregation, Exons, Family, Female, Fibromatosis, Gingival, Genetic Predisposition to Disease, Humans, Male, Middle Aged, Mutation, Pedigree, Repressor Proteins |
Abstract | Hereditary gingival fibromatosis (HGF) is the most common genetic form of gingival fibromatosis that develops as a slowly progressive, benign, localized or generalized enlargement of keratinized gingiva. HGF is a genetically heterogeneous disorder and can be transmitted either as an autosomal-dominant or autosomal-recessive trait or appear sporadically. To date, four loci (2p22.1, 2p23.3-p22.3, 5q13-q22, and 11p15) have been mapped to autosomes and one gene (SOS1) has been associated with the HGF trait observed to segregate in a dominant inheritance pattern. Here we report 11 individuals with HGF from three unrelated families. Whole-exome sequencing (WES) revealed three different truncating mutations including two frameshifts and one nonsense variant in RE1-silencing transcription factor (REST) in the probands from all families and further genetic and genomic analyses confirmed the WES-identified findings. REST is a transcriptional repressor that is expressed throughout the body; it has different roles in different cellular contexts, such as oncogenic and tumor-suppressor functions and hematopoietic and cardiac differentiation. Here we show the consequences of germline final-exon-truncating mutations in REST for organismal development and the association with the HGF phenotype. |
DOI | 10.1016/j.ajhg.2017.06.006 |
Alternate Journal | Am J Hum Genet |
PubMed ID | 28686854 |
PubMed Central ID | PMC5501868 |
Grant List | R01 NS058529 / NS / NINDS NIH HHS / United States T32 GM008307 / GM / NIGMS NIH HHS / United States U54 HG003273 / HG / NHGRI NIH HHS / United States UM1 HG006542 / HG / NHGRI NIH HHS / United States |
REST Final-Exon-Truncating Mutations Cause Hereditary Gingival Fibromatosis.
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